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By Ronald R. Watson (auth.), Ronald R. Watson (eds.)

ISBN-10: 1461202434

ISBN-13: 9781461202431

ISBN-10: 1461266785

ISBN-13: 9781461266785

Alcoholism, the oldest addictive affliction, is still a significant challenge as a result of its significant adjustments of many physiological structures. The genetic and neurological origins and mechanisms are advanced and simply partially understood. even if, it really is transparent that alcohol use modifies the creation of such a lot hormones, at once and not directly modulating the structures that hormones keep an eye on. Acute and protracted alcohol intake impacts almost all mobile capabilities of tissues like mind, liver, immune method, and reproductive and endocrine organs. The latter have significant regula­ tory roles, affecting many features from improvement to repro­ duction. even though pressure can extra alter alcohol's results on hormone construction, it considerably reasons such a lot of alcohol's modi­ fications of the physique to take place through alterations in hormone creation and secretion. Ronald R. Watson vii members Michael L. Adams • division of Psychiatry, Washington college institution of drugs, St. Louis, MO Robert A. Anderson, Jr .• Ob/Gyn learn, Rush-Presbyterian-St. Luke's scientific middle, Chicago,IL Kasibhatla Bhavani • Molecular Heptology Laboratory, Massachusetts common clinic melanoma middle, Harvard scientific tuition, Charlestown, MA Theodore J. Cicero • division of Psychiatry, Washington college college of medication, St.

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The mechanisms underlying the fact that morphine decreases LH at a very early age, whereas naloxone is ineffective in increasing LH until sexual maturation, are presently unknown. However, two explanations seem plausible: First, the Eap ligand for suppressing LH might be absent until the time of puberty and, consequently, naloxone cannot displace it from its receptor, thereby evoking a release of LH, or, second, naloxone and morphine influence LH by independent mechanisms and/or receptors. 44 Since ~-endorphin is believed to be the primary Eap involved in suppressing the release of LH-releasing hormone (LHRH) from the hypothalamus,'-6 and possibly exerting paracrine and autocrine effects on the testes,45-48 these data are consistent with the interpretation that the agedependent effects of naloxone on LH may be the result of maturational factors associated with EaP-mediated control of reproductive endocrinology.

1. Cicero (1980) Effects of exogenous and endogenous opiates on the hypothalamic-pituitary-gonadal axis in the male rat. Fed. Proc. 39,2551-2554. 2T. J. Cicero (1984) Opioid-mediated control of LH in the male: physiological implications, in Opioid Modulation of Endocrine Function, G. Delitala, M. Motta, and M. Serio, eds. Raven, New York, pp . 211-222. 'Cicero, T. J. (1987) Basic endocrine pharmacology of opioid agonists and antagonists, in Pharmacology and Clinical Uses of Inhibitors of Hormone Secretion and Action.

1 and K receptors are involved in endocrine function or whether the design of the studies masked the fact that only one receptor was actually involved. 1 and K receptors in regulating endocrine activity. Several strategies to resolve this dilemma are discussed below. 1 and K receptors in endocrine function and to permit determinations of whether either of these receptors is functionally relevant in the neuronal control of endocrine activity. These strategies deserve some explanation. Activation of Endocrine Axes Most previous studies have examined the effects of opiates on basal hormone secretion rates .

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Alcohol and Hormones by Ronald R. Watson (auth.), Ronald R. Watson (eds.)


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