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By Michael F. O’Rourke (auth.), M. E. Safar, G. M. London, A. Ch. Simon, Y. A. Weiss (eds.)

ISBN-10: 9400933037

ISBN-13: 9789400933033

ISBN-10: 9401079838

ISBN-13: 9789401079839

The hemodynamic mechanisms of high blood pressure are usually constrained to the learn of 3 dominant parameters: blood strain, cardiac output and vascular resis­ tance. consequently, the improvement of high blood pressure is generally analyzed by way of a 'struggle' among cardiac output and vascular resistance, leading to the classical development of standard cardiac output and elevated vascular resistance, therefore indicating a discount within the quality of small arteries. even if, up to now years, the medical administration of high blood pressure has mostly converted those easy perspectives. whereas an sufficient regulate of blood strain should be acquired with antihypertensive medications, arterial problems may perhaps happen, concerning more often than not the coronary circulate and suggesting that numerous elements of the cardiovascular procedure are altered in high blood pressure. certainly, disturbances within the arterial and the venous method had already been spotted in animal high blood pressure. the elemental assumption during this publication is that the general cardiovascular process is fascinated with the mechanisms of the increased blood strain in sufferers with high blood pressure: not just the guts and small arteries, but in addition the big arteries and the venous procedure. as a result, the next issues are emphasised. First, the cardiovascular procedure in high blood pressure has to be studied not just when it comes to regular stream but additionally by way of making an allowance for the pulsatile elements of the guts and the arterial platforms. moment, arterial and venous compliances are altered in high blood pressure and possibly replicate intrinsic adjustments of the vascular wall.

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7. Krieger EM, Marseillan RF (1966): Neural control in experimental hypertension: The role of baroreceptors and splanchnic fibers. Acta Physiol Lat Am 16: 343-352. 8. Aars H (1968): Aortic baroreceptor activity in normal and hypertensive rabbits. Acta Physiol Scand 72: 298-309. 9. Nosaka S, Okamoto K (1970): Modified characteristics of the aortic baroreceptor activities in the spontaneously hypertensive rat. Jpn Circ J 34: 685-693. 10. Krieger EM, Salgado HC, Michelini LC (1982): Resetting of the baroreceptors.

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Arterial and Venous Systems in Essential Hypertension by Michael F. O’Rourke (auth.), M. E. Safar, G. M. London, A. Ch. Simon, Y. A. Weiss (eds.)

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